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2 edition of morphological and chemical study of articular cartilage in pyrophosphate arthropathy (chondrocalcinosis articularis or CPPD crystal deposition disease). found in the catalog.

morphological and chemical study of articular cartilage in pyrophosphate arthropathy (chondrocalcinosis articularis or CPPD crystal deposition disease).

Anders Bjelle

morphological and chemical study of articular cartilage in pyrophosphate arthropathy (chondrocalcinosis articularis or CPPD crystal deposition disease).

by Anders Bjelle

  • 355 Want to read
  • 30 Currently reading

Published in Lund .
Written in English

    Subjects:
  • Chondrocalcinosis.

  • Classifications
    LC ClassificationsRC935.C47 B53
    The Physical Object
    Pagination34 p.
    Number of Pages34
    ID Numbers
    Open LibraryOL5249004M
    LC Control Number75321350

    Calcium pyrophosphate dihydrate crystal arthropathy preferentially affects large joints and intervertebral discs (Pritzker et al., ).Gross examination demonstrates the crystals as punctate white deposits within the articular tissues (Figure ).This contrasts with gout in which the crystals aggregate as a white paste on the articular surfaces. Bjelle AO. Morphological study of articular cartilage in pyrophosphate arthropathy. (Chondrocalcinosis articularis or calcium pyrophosphate dihydrate crystal deposition diseases). Ann Rheum Dis. Nov; 31 (6)– [PMC free article] Bjelle A, Sundén G. Pyrophosphate arthropathy: a clinical study of fifty cases.

    Request PDF | Calcium Pyrophosphate Dihydrate Deposition Disease | Development and deposition of amorphous or crystalline inorganic phases, a process referred to as mineralization, occur in a. Request PDF | Calcium pyrophosphate dihydrate crystal deposition disease: Imaging perspectives | Calcium pyrophosphate dihydrate (CPPD) crystal deposition disease is widespread in elderly persons.

    Robert Terkeltaub, in Kelley's Textbook of Rheumatology (Ninth Edition), Dysregulated Inorganic Pyrophosphate Metabolism in Pathologic Articular Cartilage Calcification. PP i is a potent inhibitor of the nucleation and propagation of BCP crystals. 19 Concordantly, maintenance of physiologic extracellular PP i levels by chondrocytes and certain other cells serves to suppress calcification. Ultrastructural studies ofpyrophosphate crystal deposition in articular cartilage S. Y. ALI,' S. GRIFFITHS,1 M. T. BAYLISS,' ANDP. A. DIEPPE2 Fromthe 'Institute ofOrthopaedics, RoyalNational OrthopaedicHospital, Stanmore, Middlesex, and2BristolRoyal Infirmary, Bristol The mechanism of calcium pyrophosphate crystal formation in the articular.


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Morphological and chemical study of articular cartilage in pyrophosphate arthropathy (chondrocalcinosis articularis or CPPD crystal deposition disease) by Anders Bjelle Download PDF EPUB FB2

Morphological study of articular cartilage in pyrophosphate arthropathy. (Chondrocalcinosis articularis or calcium pyrophosphate dihydrate crystal deposition diseases). Fallet G. Inorganic pyrophosphate in plasma, urine, and synovial fluid of patients with pyrophosphate arthropathy (chondrocalcinosis or pseudogout).

Lancet. Oct 31; 2 Cited by: 1. Ann Rheum Dis. Nov;31(6) Morphological study of articular cartilage in pyrophosphate arthropathy. (Chondrocalcinosis articularis or calcium pyrophosphate dihydrate crystal deposition diseases).Cited by:   An ultrastructural study of articular cartilage from five patients with calcium pyrophosphate dihydrate (CPPD) crystal deposition disease was performed.

The CPPD crystals, identified by micro X-ray diffraction, were usually found in clusters, located in intercellular areas of the intermediate cartilage by:   This chapter provides an overview of tissues unique to synovial joints, articular cartilage, and meniscus. The development and cellular and (bio)chemical composition are described, as well as the role of mechanical stimuli.

In addition, the role of growth factors in cartilage and meniscus homeostasis, cellular differentiation, and chondrocyte hypertrophy are discussed. An ochronotic femoral head has been studied morphologically under the light and the electron microscope.

Its articular cartilage showed the alterations already reported in the literature, mainly consisting of erosions of the surface, pigment accumulation in chondrocytes and intercellular matrix, chondrocyte degeneration, the formation of pigmented, calcified and uncalcified microshards, Cited by:   Three‐dimensional (3D) double echo stead‐state (DESS) MRI utilized for the assessment of articular cartilage morphology.

The data were acquired in sagittal orientation (A) and was reoriented into coronal (B) and axial (C) datasets for segmentation of articular cartilage.

In‐plane resolution: × mm. Slice thickness: mm. In the present study we showed that there is a significant correlation between clinical symptoms and the amount of mineralized articular cartilage. Mitsuyama et al (10) recently showed that age, rather than the presence of OA, is the predominant factor driving progressive pathologic calcification in articular cartilage.

Osteoarthritis (OA) is the commonest condition to affect synovial joints, but although any synovial joint can be affected, most studies of pathology relate to large joints (knees and hips).

OA involves the whole joint and pathological alterations typically occur in all joint tissues. Established OA is characterized by a mixture of tissue loss and new tissue production resulting in focal loss.

Scanning electron microscopy, in conjunction with freeze-fracturing and freeze-drying preparation techniques, was used to characterize the morphology and distribution of crystal deposits foundin situ in the articular cartilage of threepost mortem human knee joints.

Energy dispersive analysis and X-ray diffraction were used to analyse the chemical composition of the individual crystals.

Okazaki T, Saito T, Mitomo T, et al: Pseudogout: Clinical observation and chemical analyses of deposits. Arthritis Rheum [SuppI]Bjelle AO: Morphological study of articular cartilage in pyrophosphate arthropathy (chrondrocalcinosis articu- laris or calcium pyrophosphate dihydrate crystal deposition disease).

Bjelle, A. O.: Morphological study of articular cartilage in pyrophosphate arthropathy (Chondrocalcinosis articularis or calcium pyrophosphate dihydrate crystal deposition disease).

Ann. Rheum. Dis, – (). PubMed Google Scholar. The present study represents the first complete ultrastructural investigation of the glenoid articular osteo‐chondral complex in post‐traumatic shoulder instability, evaluating in detail cell features throughout the articular cartilage and sub‐chondral bone that is the morphological prerequisite to understand bio‐molecular mechanisms of.

Morphological Study of Articular Cartilage in Gout and Pseudogout, GeriatricsPyrophosphate Arthropathy (Chondrocalcinosis Articularis or 4.

McCarty, D. Cartilage matrix in hereditary pyrophosphate arthropathy. Bjelle A. Morphological and biochemical studies of articular cartilage were performed in 6 members of 3 families with hereditary pyrophosphate arthropathy. Evidence of metabolic disturbance of cartilage matrix was obtained from light and electron microscopic findings and by the content and composition of glycosaminoglycans.

Author(s): Bjelle,Anders Title(s): A morphological and chemical study of articular cartilage in pyrophosphate arthropathy: (chondrocalcinosis articularis or CPPD crystal deposition disease)/ by Anders Bjelle.

The therapeutic effects of bovine articular cartilage on adjuvant-induced arthritis in Kunming mice were investigated in the present study. Animals were divided into control group, model group and treatment groups (bovine articular cartilage, polysaccharide from bovine articular cartilage, polysaccharide from bovine laryngeal cartilage; administered orally by gastric intubations, mg/kg/d.

In both diseases, there is mineralization of the articular cartilage and menisci, synovial hypertrophy, and cartilage thinning. Most patients also have OA (1, 2).

It is possible that the increased mineralization observed in humans with chondrocalcinosis and BCP crystal deposition disease could result in altered joint biomechanics, ultimately.

Articular cartilage in the degenerative arthropathy of hemochromatosis Articular cartilage in the degenerative arthropathy of hemochromatosis Schumacher, H.

Ralph Iron was readily demonstrated in chondrocytes in 3 of 4 hemochromatosis articular cartilages studied. Either apatite, calcium pyrophosphate dihydrate crystals, or both were found in all cartilages including those.

Morphological analysis of articular cartilage biopsies from a randomized, clinical study comparing the effects of – kDa sodium OA and rheumatoid arthritis (RA) the molecular weight of we carried out a histological and electron microscopy study on cartilage samples taken from osteoarthritic human.

Objective: The purpose of this case-study was to perform morphological and molecular analysis of articular cartilage biopsies from the femoral condyle of a 33 year old woman with intra-articular.

Cartilage has been studied from 12 patients with different degrees of chondrocalcinosis. Degenerative changes and patches of decreased saphranin 0 staining were present even in areas without calcium deposits. Alkaline phosphatase was identified only in the basal layer and was slightly decreased.

By electron microscopy mildly involved cartilage showed small diameter isolated crystals lying in a.Morphological Study: Ultrastructural Aspects of Articular Cartilage and Subchondral Bone in Patients Affected by Post‐Traumatic Shoulder Instability Paolo Baudi Dipartimento di Chirurgia Ortopedica, Azienda Ospedaliero‐Universitaria Policlinico di Modena, Università di Modena e R.E, Modena, Italy.The purpose of this study was to investigate age-related changes in the morphological, biochemical and mechanical properties of articular cartilage (AC) and subchondral bone in the rat tibial plateau.